Studies in cardiac pathology . of Abbotts cases it appeared incombination with other defects; most commonly with pulmonarystenosis. When it occurs alone, it produces hypertrophy of bothventricles, the degree of which is dependent upon the size of theperforation. As the right ventricle hypertrophies and pressurein the right ventricle increases, the leakage diminishes, so that theeffect of the lesion tends to correct itself; on the other hand, thepressure in the pulmonary artery increases. But since the ordinaryresistance in the pulmonary circulation is much less than in thesystemic, when the fo
Studies in cardiac pathology . of Abbotts cases it appeared incombination with other defects; most commonly with pulmonarystenosis. When it occurs alone, it produces hypertrophy of bothventricles, the degree of which is dependent upon the size of theperforation. As the right ventricle hypertrophies and pressurein the right ventricle increases, the leakage diminishes, so that theeffect of the lesion tends to correct itself; on the other hand, thepressure in the pulmonary artery increases. But since the ordinaryresistance in the pulmonary circulation is much less than in thesystemic, when the forces of both ventricles approximate oneanother the effect on the pulmonarj^ circulation is the same asthough the left ventricle became weaker and the right remainedunchanged. Pulmonaiy engorgements may, therefore, result,with consequent dyspnea. In most cases, however, the hyper-trophy does not reach this point, and it is only when the heart isstimulated by effort or exercise that pulmonary engorgement setsin. (Hirschfelder.). Fig. 75.—Patent Ductus Arteriosus. Male infant, thirty-four days old. (Physician: Dr. Pathologist: Dr. A. ) Clinical Notes: At birth the baby was cyanotic and weighed 7 pounds. Respiratorydifficulty was present at all times, and the cyanosis was marked during the first few the blueness became less prominent, especially during short periods in which it almostdisappeared. Death occurred during an attack of dyspnea associated with cyanosis. Pathologic Diagnosis: Transposilion of the aorta and the pulmonary artery; patentdiictics arteriosus; hypertrophy of the right ventricle. Patent foramen ovale; partial atelectasisof the right lung; general visceral congestion. Pathologic Notes: The heart is essentially normal in size. The wall of the right andleft ventricles are respectively 5 and 7 mm. in thickness. Neither the pulmonary artery northe aorta shows any evidence of stenosis. The foramen ovale is patulous in the form of aslit-l
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Keywords: ., bookauthornorrisgeorgewilliam18, bookcentury1900, bookdecade1910
