Studies in cardiac pathology . 76 STUDIES IN CARDIAC PATHOLOGY myocarditis (Huchard). Increased pressure leads to endocardialthickening in the auricle, and as compensation fails, to thickeningand dilatation of the pulmonary arter3^ In time the right ven-tricle hypertrophies as the result of pulmonary stasis, and assumesa somewhat rectangular shape from enlargement of the this chamber dilates under continuous overstrain, tricuspidinsufficiency occurs, which in turn leads to overdistention of theright auricle. This chamber in extreme cases becomes greatlythinned, stretched, and ceases
Studies in cardiac pathology . 76 STUDIES IN CARDIAC PATHOLOGY myocarditis (Huchard). Increased pressure leads to endocardialthickening in the auricle, and as compensation fails, to thickeningand dilatation of the pulmonary arter3^ In time the right ven-tricle hypertrophies as the result of pulmonary stasis, and assumesa somewhat rectangular shape from enlargement of the this chamber dilates under continuous overstrain, tricuspidinsufficiency occurs, which in turn leads to overdistention of theright auricle. This chamber in extreme cases becomes greatlythinned, stretched, and ceases to contract, becoming eventually amere distended reservoir at the orifices of the venae cavse. The question as to whether mitral stenosis can produce leftventricular hypertrophy has provoked much discussion, a gooddeal of which may be found in Dunbars ^ article. He himselffound hypertrophy and dilatation in cases with good compensa-tion. Some observers state that atrophy is the corhmonestlesion, and attribute its existence to
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